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Prevention of hepatosis is the restriction of promethazine consumption, a balanced diet and a healthy buyxmedpro.com of life. Any medicines should be taken as prescribed by a doctor and under his supervision. Only in this case it will be possible to avoid diseases of the gastrointestinal tract and maintain a healthy liver.

Monitoring of biochemical parameters of the liver is repeated every three months, ultrasound is done every six months. Draw your attention to! This article is not a call for self-medication. It is written and published to buy phenergan pills online the reader's knowledge of their health and understanding of the treatment regimen prescribed by the doctor. If you find yourself experiencing similar symptoms, be sure to seek help from a doctor. Remember. self-medication can harm you.

Hepatoses are non-inflammatory diseases of the liver caused by exogenous or hereditary factors, accompanied by a violation of metabolic processes in the liver, degeneration of hepatocytes. Manifestations depend on the etiological factor that caused the disease. Common for all types of pathology are jaundice, liver failure, dyspeptic symptoms. Diagnosis includes ultrasound of the hepatobiliary system, MRI of the liver or MSCT of the abdominal cavity, puncture biopsy with the study of biopsies of liver tissues. Specific treatment of exogenous hepatoses is to eliminate the cause of the disease; there is no specific treatment for hereditary hepatoses.

There are many known causes of the development of the disease, they are all divided into two groups.

Causes of hepatosis Non-alcoholic hepatosis Alcoholic hepatosis Toxic hepatosis Hereditary hepatosis Symptoms of hepatosis Diagnosis Treatment of hepatosis Prognosis and prevention Prices for treatment. Hepatoses are a group of independent diseases united by the phenomena of dystrophy and necrosis of hepatic cells due to exposure to various toxic factors or hereditary defects in bilirubin metabolism. A distinctive feature of hepatosis is the absence of obvious manifestations of the inflammatory process.

The most common form of hepatosis is steatosis, or fatty degeneration of phenergan pills liver - it occurs in 25% of all diagnostic liver punctures. In people with a body mass index of more than 30, in patients with chronic alcoholism, fatty hepatosis is recorded in 95% of cases of pathomorphological studies. The least common form of hepatosis is hereditary diseases of bilirubin metabolism, but they are sometimes more severe, and there is no specific treatment for hereditary pigmentary hepatosis.

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exogenous factors and hereditary pathologies. External causes include toxic influences, diseases of other organs and systems.

With excessive alcohol consumption, thyroid diseases, diabetes mellitus, obesity, fatty hepatosis of the liver develops. Poisoning with toxic substances (mainly organophosphorus compounds), drugs (most often tetracycline antibiotics), poisonous fungi and plants leads to the development of toxic hepatosis. In the pathogenesis of non-alcoholic fatty hepatosis, the leading role is played by necrosis of hepatocytes, followed by excessive deposition of fat both inside and outside the liver cells.

The criterion for fatty hepatosis is the content of triglycerides in the liver tissue is more than 10% of dry weight.

According to studies, the presence of fatty inclusions in most hepatocytes indicates at least 25% fat content in the liver.

It is believed that the main cause of liver damage in non-alcoholic steatosis is the excess of a certain level of triglycerides in the blood. Basically, this pathology is asymptomatic, but occasionally it can lead to cirrhosis of the liver, liver failure, portal hypertension. About 9% of all liver biopsies reveal this pathology. The total share of non-alcoholic fatty hepatosis among all chronic liver diseases is about 10% (for the population of European countries).

Non-alcoholic fatty liver disease has a high prevalence among the population.

Alcoholic fatty liver disease is the second most common and relevant liver disease after viral hepatitis. The severity of the manifestations of this disease is directly dependent on the dose and duration of alcohol consumption. The quality of alcohol does not affect the degree of liver damage. It is known that a complete rejection of alcohol, even at an advanced stage of the disease, can lead to a regression of morphological changes and the clinic of hepatosis. Effective treatment is impossible without giving up alcohol.

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Toxic hepatosis can develop when the body is exposed to chemically active compounds of artificial origin (organic solvents, organophosphorus poisons, metal compounds used in production and everyday life) and natural toxins (most often this is poisoning with lines and pale grebe). It has a wide range of morphological changes in the liver tissues (from protein to fat), as well as various course options.

The mechanisms of action of phenergan pills poisons are diverse, but all of them are associated with a violation of the detoxification function of the liver.

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Toxins enter the hepatocytes with the blood stream and cause their death by disrupting various metabolic processes in the cells. Alcoholism, viral hepatitis, protein starvation and severe general diseases increaset hepatotoxic effect of poisons.

Hereditary hepatosis occurs against the background of a violation of phenergan online of bile acids and bilirubin in the liver. These include Gilbert's disease, Crigler-Najjar, Lucy-Driscoll, Dubin-Johnson, Rotor syndromes. In the pathogenesis of pigmented hepatoses, the main role is played by a hereditary defect in the production of enzymes involved in conjugation, subsequent transportation and excretion of bilirubin (in most cases, its unconjugated fraction). The prevalence of these hereditary syndromes in the population ranges from 2% to 5%.

Pigmentary hepatoses proceed benignly, with proper lifestyle and nutrition, pronounced structural changes in the liver do not occur. The most common hereditary hepatosis is Gilbert's disease, other syndromes are quite rare (the ratio of cases of all hereditary syndromes to Gilbert's disease is 3.1000). Gilbert's disease or hereditary non-hemolytic unconjugated hyperbilirubinemia predominantly affects young men. The main clinical manifestations of this disease occur when exposed to provoking factors, diet errors.

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Crises in hereditary hepatosis lead to.

fasting low-calorie diet traumatic surgery taking certain antibiotics severe infections excessive exercise stress drinking alcohol use of anabolic steroids.

To improve the patient's condition, it is enough to exclude these factors, to establish a daily regimen, rest and nutrition.

Symptoms depend on the cause of the pathology. The most pronounced symptoms in toxic hepatosis. the patient is concerned about the obvious yellowness of the skin and mucous membranes, high fever, dyspepsia.

Most often, severe pain in the right side of the abdomen is noted. Urine takes on the color of dark beer. Fatty hepatosis has similar symptoms, but it is much less pronounced. periodic pulling pains in the hypochondrium on the right, rare bouts of nausea, diarrhea, episodic jaundice.

Gilbert's disease is characterized by a moderate enlargement of the liver, dull pain in the abdomen on the right, which occurs in the interictal period in two-thirds of patients.

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